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Genetic knockout and pharmacological blockade of both Aldh1a1 and GAT1 have been reported to reduce GABAergic inhibitory post-synaptic currents (IPSCs) in striatal DA/GABA neurons (Tritsch et al., 2014

[14] Whether those effects have been independently replicated across groups to the standard the word reproducible implies is a separate question, and one this article is deliberately not asserting

pH and stability GHK-Cu maintains stability within specific pH ranges

KEAP1 belongs to the BTB-Kelch family of proteins, known to be involved in ubiquitination [116, 117]